CheLiu

The measles virus is single-stranded, negative-sense RNA genome. The genome is about 15,900 nucleotides long.

RNA itself can’t function as a virus. It need viral proteins (already inactivated during the vaccine preparation) to function as a virus.

There is a sonication, which breakdown the RNA, during MMR vaccine preparation.

The study use PCR, which only detect small fragments of the viral RNA. There is no evident of shedding of the RNA genome.

This is the Yale scientist

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Drop a ??if you’re not going to comply again.

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HKU5 is a Pipistrellus bat coronavirus that discovered by Hong-Kong University (HKU) long time ago. The virus can’t interact with or amplify in human cells. The new HKU5-CoV-2 has spike protein that can interact with human cell receptor ACE2 (with low efficiency). Coronaviruses contain 28~32 proteins. They need almost all their ~30 proteins to be able to function in human cells to be amplified in human cells. Even the spike protein can be Sars-CoV-2 like (ACE2 binding) the rest of HKU5-CoV-2 proteins remain HKU5-like. Coronavirus has very high mutation rate. If HKU5 continually cross infects between human and bat, it may eventually generate enough adaptive mutations allowing it to grow in human cells. That’s why when a new virus jump from other species to human, you want to prevent the spread at early stage to avoid it eventually well adapt to human after enough time in human (like SARS-CoV-2). I am more concerned about other SARS-CoV-1 or 2-like bat coronaviruses that already have most of their other proteins potentially adapted to human functions. Once their spikes adapt to human receptors (not necessary ACE2, other human cell receptors also work), they will be problems.

T-cell exhaustion is an epigenetic reprogramming that differ by individual.

It is well known that unvaccinated patients suffer significant T-cell exhaustion than vaccinated patients (about 9000 publications if you are interested LINK =) after COVID.

Covid vaccines cause some T-cell exhaustion (as well as many other infections diseases) but not statistic significant.

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To be nice, your posts show you have no knowledge or understanding of immunity and the history of vaccines. To be blunt, you are cementing yourself as a dumbass with your continued posts about this subject.

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To provide direct prevention of respiratory virus infection, you need mucosal immunity from vaccines that target the mucosal tissues of the gastrointestinal, pulmonary, and genitourinary tracts, aiming to induce localized immune responses at these sites, primarily by generating secretory IgA antibodies, to prevent infections that enter the body through these mucosal surfaces. Currently, most vaccines are primarily stimulate blood-based antibodies that neutralize pathogens that already entered.

High school biology

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Stopped right there. Vaccines prevent infection. I don’t know anyone who got those shots and DIDN’T become infected.

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Vaccines prepare body’s ability to produce antibodies after infection and reduce severity, not prevent infection. Mask wearing is the one reduce the chance of infection

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But compare that number of people who die of the flu or pneumonia. Unfortunately I had Covid and pneumonia at the same time and I still don’t have the ability to smell. I believe at the time flu deaths and pneumonia deaths went down iirc

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Covid causes pneumonia

Wearing a mask can help lower the risk of respiratory virus transmission, including flu, respiratory syncytial virus (RSV), and COVID-19 (SARS-COV-1&2)

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Confirmed how?

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Clinical report (by doctor) plus lab report. Usually will have 5~7 day delay